Possible therapy for ALS based on the cyanobacteria/BMAA hypothesis

Research output: Contribution to journalArticle

5 Scopus citations

Abstract

Although the cyanobacteria/BMAA hypothesis of the cause of ALS and other age-related neurodegenerative diseases remains to be proven, it is not too early to ask whether treatment would be possible if the hypothesis were correct. This paper reviews the possible ways that chronic BMAA neurotoxicity could be prevented or treated.

Original languageEnglish (US)
Pages (from-to)118-123
Number of pages6
JournalAmyotrophic Lateral Sclerosis
Volume10
Issue numberSUPPL. 2
DOIs
StatePublished - Dec 1 2009

Keywords

  • Aminoacyl-tRNA synthetase
  • Amyotrophic lateral sclerosis (ALS)
  • Blood-brain barrier
  • BMAA
  • Therapeutic trial

ASJC Scopus subject areas

  • Clinical Neurology
  • Neurology

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