The pathogenesis of benign peptic ulcer disease, both gastric and duodenal, remains inknown. Because neither form occurs in the absence of acid (and the majority of duodenal ulcer patients exhibit hypersecretion of acid), investigations over decades have sought to clarify the mechanisms responsible for the control of parietal cell function.
|Title of host publication||COMPR.THER.|
|Number of pages||9|
|State||Published - Dec 1 1976|
ASJC Scopus subject areas