A patient with polymyositis had the pathological hallmarks of inclusion body myositis (IBM) as well as features that to our knowledge have never before been reported in IBM - an associated collagen-vascular disease (Sjogren's syndrome) and tubuloreticular structures in capillary endothelial cells. The pathogenesis of IBM is unknown, but its coexistence with Sjogren's syndrome suggests that immune mechanisms may play a role in producing muscle damage.
|Original language||English (US)|
|Number of pages||3|
|Journal||Archives of neurology|
|State||Published - Mar 1982|
ASJC Scopus subject areas
- Arts and Humanities (miscellaneous)
- Clinical Neurology