Aponeurosis Disinsertion in Congenital Entropion

David T. Tse, Richard L. Anderson, Jonathan D. Fratkin

Research output: Contribution to journalArticle

25 Scopus citations

Abstract

Lower lid retractor aponeurosis disinsertion is a well-recognized etiologic factor in many involutional entropion cases, but to our knowledge it has not previously been reported as a cause of congenital entropion. Four congenitally entropic lower eyelids in three patients with no history of birth trauma were all found to have retractor disinsertion during exploratory procedures. All four eyelids were surgically corrected by reinserting the retractors to the inferior tarsal margin. Detailed histologic studies of orbicularis oculi muscle fibers in two cases showed no evidence of fiber hypertrophy. This finding refutes the commonly accepted concept of orbicularis muscle hypertrophy as an etiologic mechanism of congenital entropion.

Original languageEnglish (US)
Pages (from-to)436-440
Number of pages5
JournalArchives of ophthalmology
Volume101
Issue number3
DOIs
StatePublished - Mar 1983
Externally publishedYes

ASJC Scopus subject areas

  • Ophthalmology

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