Abstract
An early manifestation of the response of WRK-1 rat mammary tumor cells to vasopressin is an increase in incorporation of (32P)Pi into phospholipids. Incorporation into all classes of phospholipids is stimulated; however, incorporation into phosphatidylinositol (Pl) is increased to the greatest degree (3- to 10-fold as compared with 1.3- to 2-fold for the other phospholipids). Furthermore, increased incorporation into Pl is accompanied by an increased rate of Pl turnover; turnover rates of the other phospholipids are unaffected by vasopressin.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 148-153 |
| Number of pages | 6 |
| Journal | Journal of Cellular Physiology |
| Volume | 112 |
| Issue number | 1 |
| DOIs | |
| State | Published - Jul 1982 |
ASJC Scopus subject areas
- Physiology
- Clinical Biochemistry
- Cell Biology
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Cite this
Monaco, M. E., & Lippman, M. E. (1982). A new model system for studying the phosphatidylinositol cycle. Journal of Cellular Physiology, 112(1), 148-153. https://doi.org/10.1002/jcp.1041120122